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For example, a dysfunctional endothelium can be associated with changes of endothelial cell phenotypes with increased proliferation, anoikis, altered morphology, production of C-reactive protein, and other inflammatory and thrombogenic mediators, including monocyte chemotactic protein-1 and plasminogen activator inhibitor-1, upregulated adhesion molecules, and enhanced thrombogenicity and adhesiveness for circulating cells (Safar et al., 2003)