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The neurotoxic effects of mHtt are mediated by the induction of mitochondrial defects, which in turn result in aberrant energy metabolism and HD-related neuronal dysfunction ( Studies have demonstrated that mHTT interacts with the PGC-1α promoter, directly impairs the activity of PGC-1α, and impedes the capacity of PGC-1α to open downstream target genes (Sharma A

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After a single intravenous (IV) administration, single intramuscular (IM) administrations at three doses in successive increments along with repeated IM administrations, the elimination half-life (t 1/2 ) of prototype BPC157 was less than 30 min, and BPC157 showed linear pharmacokinetic characteristics in rats and beagle dogs at all doses